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On the contrary, CCl 4 mice showed approximately 3. In contrast, when carbon tetrachloride was administered to Tc-inoculated subjects, a scarce infiltration of polymorphonuclear leukocytes was denoted at the hepatic parenchyma of these animals Fig.
To this respect, S. Early exposure of infants to GI nematodes induces Th2 dominant immune responses which are unaffected by periodic anthelminthic treatment.
A AGUAYO HERMOZA, PAOLA ALEJANDRA. ECONOMIA. D CABRERA QUISPE, DULY SHARAT. K CHAMPI CHAVEZ, KARINA KAREN. F ESCOBEDO BACA, DAPHNE. Alexander Demkov, University of Texas at Austin, USA. Sergei Dudarev José María Cabrera Marrero, Universitat Politècnica de Catalunya, Spain Alma Gabriela Palestino-Escobedo, Universidad Autónoma de San Luis Potosí, Mexico Karina Suarez Alcantara, Instituto de Investigaciones en Materiales, Mexico.
Tc-inoculated and non-inoculated mice orally received carbon tetrachloride or olive oil twice per week during six weeks.
All results from C to J are presented as optical densitometry OD using 18S as control gene of constitutive expression. Food Chem Toxicol. Higuchi H, Gores GJ. Scand J Immunol. Th2-biased cytokine profile induces a hepatic microenvironment consistent with alternative macrophage activation. Click on the image to enlarge.
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|In contrast, carbon tetrachloride-induced liver fibrosis was significantly less in non-inoculated animals than in the Tc-inoculated group.
Furthermore, the hepatic tissue from Tc-inoculated animals displayed a significant fold increase in Ym-1 expression Fig. Thus, the evaluation of whether the presence of helminth parasites correlates with severe liver fibrosis in a more real clinical scenario is worthy of consideration, since these pathogens have exerted the ability to switch the host immune response from the Th1-profile toward a Th2-type in both mice and humans [ 56 - 58 ].
Kupffer cells in non-alcoholic fatty liver disease: the emerging view. This procedure was repeated 3 times using non-supplemented sterile PBS 1X. Independent variables were presence or absence of inoculation with T.